But error-prone also.

Using an animal model, the scientists then investigated the consequences of a defective switch on cardiac development. Embryonic development can be investigated particularly efficiently in the zebrafish. The Bad Nauheim-based researchers consequently produced a genetically modified seafood that lacked a functioning Ajuba protein. Cardiac development in these fishes was in fact severely disrupted. In addition to deformation of the heart, caused by twisting of the cardiac axis, what particularly struck the experts was a notable difference in size in comparison to control animals. ‘In virtually all the investigated fish we observed a dramatic enlargement of the center. If Ajuba is usually absent, there is actually no other change that finally silences the Isl1-controlled component of cardiac advancement’, says Dobreva.This article can be republished with kind permission from our close friends at The Kaiser Family Foundation. You can view the complete Kaiser Daily Health Policy Report, search the archives, or sign up for email delivery of in-depth coverage of health policy developments, debates and discussions. The Daily Health Plan Report is released for Kaisernetwork.org, a free of charge program of The Henry J. Kaiser Family Foundation. Copyright 2009 Advisory Plank Company and Kaiser Family Foundation.